Microreviews, Review

VAMP8 mucin exocytosis attenuates intestinal pathogenesis by Entamoeba histolytica

Steve Cornick1, France Moreau1, Herbert Y. Gaisano2, Kris Chadee1

This article comments on work published by Cornick et al. (mBio, 2017), which nominates SNARE-mediated exocytosis as the putative mechanism responsible for pathogen-induced mucus secretion from goblet cells.

Shutdown of interferon signaling by a viral-hijacked E3 ubiquitin ligase

Kaitlin A. Davis1 and John T. Patton2

This article comments on work published by Davis et al. (mBio, 2017), which describes molecular requirements that govern NSP1 recognition of β-TrCP, including an essential degron phosphorylation event, and the step-wise incorporation of NSP1 into hijacked cullin-RING E3 ligases (CRLs) that ubiquitinate and tag β-TrCP for degradation.

Breaking the bad: Bacillus blocks fungal virulence factors

François L. Mayer1 and James W. Kronstad1

This article comments on work published by Mayer & Kronstad (mBio, 2017), which identified the soil bacterium, Bacillus safensis as a potent inhibitor of virulence factor production by two major fungal pathogens of humans, Cryptococcus neoformans, and Candida albicans.

The integrated stress response in budding yeast lifespan extension

Spike D.L. Postnikoff1, Jay E. Johnson2 and Jessica K. Tyler1

 

This article summarizes how the budding yeast Saccharomyces cerevisiae has been instrumental in unraveling the molecular and cellular determinants of aging, and how the induction of cellular stress responses has been associated with experimental lifespan extension, thus underscoring the value of yeast as a model for developing potential aging therapies for humans.

Yeast for virus research

Richard Yuqi Zhao

This article summarizes the use of budding yeast (Saccharomyces cerevisiae) and fission yeast (Schizosaccharomyces pombe) in virus research, highlighting their advantages for studying viral replication, interaction with host cells, and fundamental cellular processes affected by viruses, while discussing their potential in analyzing small viral genomes and facilitating the discovery of antiviral drugs.

Macrophages as drivers of an opportunistic infection

Annette C. Vergunst1, Nazareth Lopez Carranza1, Lili Zhang1,2, Margarida C. Gomes1, Yara Tasrini1,
Annemarie H. Meijer3 and David O’Callaghan1

This article comments on work published by Mesureur et al. (PloS Pathog, 2017), which shows that macrophages are essential for proliferation of B. cenocepacia in the host. This suggests a new paradigm for Bcc infections and urges the development of novel anti-infectious therapies to efficiently disarm these intrinsically antibiotic resistant facultative intracellular pathogens.

A yeast model for the mechanism of the Epstein-Barr virus immune evasion identifies a new therapeutic target to interfere with the virus stealthiness

María José Lista1, Rodrigo Prado Martins2, Gaelle Angrand1, Alicia Quillévéré1, Chrysoula Daskalogianni2, Cécile Voisset1, Marie-Paule Teulade-Fichou3, Robin Fåhraeus2 and Marc Blondel1

This article comments on a publication by Lista et al. (Nature Communications, 2017) that uncovered the role of the host cell nucleolin (NCL) in EBNA1 self-limitation of expression via a direct interaction of this protein with G-quadruplexes (G4) formed in GAr-encoding sequence of EBNA1 mRNA.

Exacerbating and reversing lysosomal storage diseases: from yeast to humans

Tamayanthi Rajakumar1, Andrew B. Munkacsi1,2 and Stephen L. Sturley3

This article summarizes the use of yeast models in advancing our understanding of lysosomal storage diseases (LSDs), where they have been instrumental in researching LSD mechanisms, screening for therapeutic compounds, and exploring genetic and gene-environment interactions relevant to diseases like Batten disease, cystinosis, and Niemann-Pick type C disease, as well as their connection to broader health issues such as viral infections and obesity.

Live fast, die fast principle in a single cell of fission yeast

Hidenori Nakaoka1

This article comments on a recent study (Nakaoka and Wakamoto, PLoS Biol, 2017), which developed a microfluidics-based platform to track multiple single cell lineages until death.

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Control of the gut microbiome by fecal microRNA

March 9, 2016

This article comments on work published by Liu et al. (Cell Host & Microbe, 2016), which identifies miRNAs in gut lumen and feces of both mice and humans that were able to enter bacteria, specifically regulate bacterial gene transcripts and affect bacterial growth thereby regulating the gut microbiome.

Mitochondrial regulation of cell death: a phylogenetically conserved control

February 23, 2016

Mitochondria are fundamental for eukaryotic cells as they participate in critical catabolic and anabolic pathways. Moreover, mitochondria play a key role in the signal transduction cascades that precipitate many (but not all) regulated variants of cellular demise. In this short review, the authors discuss the differential implication of mitochondria in the major forms of regulated cell death.

Mek1/Mre4 is a master regulator of meiotic recombination in budding yeast

February 22, 2016

This article comments on work published by Chen et al. (PLoS BIol, 2015), showing that the meiosis specific kinase Mek1 indirectly regulates the crossover/non-crossover decision between homologs as well as genetic interference and suggests Mek1 to be a "master regulator" of meiotic recombination in budding yeast.

Shaping meiotic chromosomes with SUMO: a feedback loop controls the assembly of the synaptonemal complex in budding yeast

February 19, 2016

This article comments on work published by Leung et al. (J Cell Biol, 2015), which shows that the formation of the meiosis-specific synaptonemal complex is controlled through SUMOylation of a regulator required for the assembly of transverse filaments, implicating the involvement of a positive feedback loop in the control of synaptonemal complex assembly.

Learning epigenetic regulation from mycobacteria

January 18, 2016

This article comments on work published by Koshla et al. (Nat Commun, 2015), which shows that pathogenic Mycobacterium tuberculosis has evolved strategies to hijack the epigenetic regulation of host transcripton for its own survival.

Location, location, location. Salmonella senses ethanolamine to gauge distinct host environments and coordinate gene expression

January 18, 2016

This article comments on work published by Anderson and Kendell (PLoS Pathog, 2015), which demonstrates that Salmonella enterica serovar Typhimurium (Salmonella) exploits ethanolamine signaling to adapt to distinct host environments to precisely coordinate expression of genes encoding metabolism and virulence.

Biofilm assembly becomes crystal clear – filamentous bacteriophage organize the Pseudomonas aeruginosa biofilm matrix into a liquid crystal

December 31, 2015

This article comments on work published by Secor et al. (Host Cell & Microbe, 2015), which highlights a previously unknown role for filamentous Pf phage in organizing the P. aeruginosa biofilm matrix into a liquid crystalline structure. These findings help ground our understanding of biofilm formation within established paradigms of soft matter physics

Histone modifications as regulators of life and death in Saccharomyces cerevisiae

December 31, 2015

The mechanism by which chromosomes restructure during apoptosis is still poorly understood, but it is becoming increasingly clear that altered epigenetic histone modifications are fundamental parameters that influence the chromatin state and the nuclear rearrangements within apoptotic cells. This review highlights recent work on the epigenetic regulation of programmed cell death in budding yeast.

Spermidine cures yeast of prions

December 25, 2015

This article comments on work published by Speldewinde and Grant (Mol Biol Cell, 2015), which found that spermidine, a polyamine that has been used to increase autophagic flux, acts as a protective agent which prevents spontaneous prion formation in yeast.

Histone deacetylases: revealing the molecular base of dimorphism in pathogenic fungi

November 4, 2015

Fungi, as every living organism, interact with the external world and have to adapt to its fluctuations. For pathogenic fungi, such interaction involves adapting to the hostile environment of their host. Survival depends on the capacity of fungi to detect and respond to external stimuli, which is achieved through a tight and efficient genetic control. Elías-Villalobos et al. propose that histone acetylation is critical to the proper timing and induction of transcription of the genes encoding factors that coordinate changes in morphology with pathogenesis.

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